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Musculoskeletal Pain: Expanded Clinical Strategies: published by the Institute for Functional Medicine: New evidence-based approaches and protocols for prototypical pain syndromes
Leading-edge treatment protocol for Fibromyalgia
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Fibromyalgia is a specific metabolic disease caused by dysbiotic mitochondropathy.  The disease is cured by addressing the underlying primary and resultant secondary problems.  This book by Dr Vasquez explains the scientific basis for the pathogenesis and treatment of migraine headaches, rheumatoid arthritis, chronic low back pain, and fibromyalgia.  

Introduction: Fibromyalgia (FM) is commonly described as an idiopathic syndrome principally characterized by widespread body pain and numerous myofascial tender points at specific locations. The condition is most common in women aged 20-50 years and often presents with associated complaints of fatigue, headaches, subjective numbness, altered sleep patterns, and gastrointestinal disturbances. FM in children and adolescents presents similarly to FM in adults except for the comparatively higher prevalence of sleep disturbance and the finding of fewer tender points.[i] Until recently FM was considered a diagnosis of exclusion after inflammation, autoimmunity, or other primary causes had been excluded by clinical and laboratory assessment. However, current criteria base the diagnosis on positive findings of chronic widespread musculoskeletal pain in the characteristic locations described later. FM shares several clinical and demographic features with chronic fatigue syndrome (CFS) and irritable bowel syndrome (IBS); the reason for these overlaps is not generally understood by most clinicians and researchers but will be made plain later in this section. The previous lack of widespread understanding about the organic nature of the illness has lead individual clinicians and authors of widely cited articles to describe FM as idiopathic, psychogenic, or neurogenic. Unfortunately, the prevailing medical view is that the condition is idiopathic with strong neuropsychogenic influences and that since the underlying causes of the condition have not been identified the best therapeutic approach is symptom suppression via perpetual pharmacotherapy with adjunctive use of psychotherapy and limited exercise.[ii],[iii],[iv]



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[i] Siegel DM, Janeway D, Baum J. Fibromyalgia syndrome in children and adolescents: clinical features at presentation and status at follow-up. Pediatrics. 1998 Mar;101(3 Pt 1):377-82

[ii] Chakrabarty S, Zoorob R. Fibromyalgia. Am Fam Physician. 2007 Jul 15;76(2):247-54

[iii] Tierney ML. McPhee SJ, Papadakis MA (eds). Current Medical Diagnosis and Treatment 2006, 45th Edition. New York: Lange Medical Books, pages 820-821

[iv] Simms RW. Nonarticular soft tissue disorders. In Andreoli TE, Carpenter CCJ, Griggs RC, and Benjamin IJ (eds). Cecil Essentials of Medicine. Seventh Edition. Philadelphia; Saunders Elsevier, 2007: 851-2
 

 

 

 

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